Abstract
In the antisaccade task, pre-cueing the location of a correct response has the paradoxical effect of increasing errors. It has been suggested that this effect occurs because participants adopt an "antisaccade task set" and treat the cue as if was a target - directing attention away from the precue and towards the location of the impending target. This hypothesis was tested using a mixed pro / antisaccade task. In addition the effects of individual differences in working memory capacity and schizotypal personality traits on performance were examined. Whilst we observed some modest relationships between these individual differences and antisaccade performance, the strongest predictor of antisaccade error rate was uncued prosaccade latency.
Highlights
In the antisaccade task participants are instructed to refrain from looking at a sudden onset target, and direct their gaze instead to its mirror image location
The task by trial type interaction was significant (F(1.4, 80) = 18.6, p < .001, r = .43, Greenhouse-Geisser correction used e = .67) reflecting the fact that errors were increased in the antisaccade task compared to the prosaccade task more in the UC and Cued Same (CS) trials compared to the CD trials
In this study we explored the effects of a number of individual differences on antisaccade performance
Summary
In the antisaccade task participants are instructed to refrain from looking at a sudden onset target, and direct their gaze instead to its mirror image location. Numerous studies have shown that, compared to healthy controls, certain psychiatric and neurological populations make an increased number of such antisaccade errors (see Hutton & Ettinger 2006; Munoz & Everling, 2004 for reviews). Increased errors in these patient groups are typically interpreted as reflecting frontally mediated impairments in inhibitory processes. Whilst there are many studies describing increased antisaccade error rates in patient populations, there has been comparatively little research into what individual differences might underlie such dramatic variations in healthy control performance
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