Abstract
Fifty mg of thioridazine in normal subjects significantly increased pain tolerance to an electric shock, and significantly reduced GSR responses to shock. Level of arousal (anxiety), as assessed by both GSR responsiveness and change in heart beat to a mild novel stimulus, was significantly reduced by thioridazine as predicted. Although the results were not entirely consistent, there was evidence that autonomic conditionability was significantly impaired by the drug. The effect of thioridazine on learning and other cortical functions has yet to be explored.
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