Abstract
In order to investigate the mechanism that high SGO-T level caused by acute myocardial infarction return to the normal level, the influence of the co-enzyme, pyridoxal phosphate (PAL-P), on SGO-T activity was studied. The changes of SGO-T activity with the addition of PAL-P in the cases with elevated SGO-T level produced experimentally was entirely different from that in the normal. In all of the former cases, a marked increase of SGO-T activity with the addition of PAL-P was recognized. In two clinical cases of acute posterior infarction, an increase of SGO-T activity to abnormal level was observed when PAL-P was added to the serum samples taken in the early stage of myocardial infarction. Therefore, measurement of the increase of SGO-T activity by adding PAL-P was considered to be useful for unveiling minimal discharge of GO-T into the blood.
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