Abstract
1. Injections of prolactin or ACTH both caused sodium retention in intact F. kansae transferred into distilled-deionized water. The response to prolactin was more pronounced and more prolonged. 2. Prolactin treatment caused a significant elevation of plasma sodium levels of hypophysectomized animals held in tap water or sodium-enriched tap water. 3. Prolactin stimulates sodium influx in dilute saline without affecting sodium efflux. ACTH stimulates both components of sodium flux. 4. Hypophysectomy reduces sodium turnover of animals held in 127% sea water. ACTH stimulates sodium turnover and causes a net reduction of total-body sodium levels. Prolactin further reduces sodium turnover and causes a marked elevation of total-body sodium. 5. Prolactin stimulates the interrenal of hypophysectomized animals held in fresh water. 6. The diameter of the interrenal nuclei is significantly less in animals adapted to 127% sea water. Both prolactin and ACTH stimulate the interrenal under such conditions.
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