Abstract
New Zealand spinach juice, when mixed with tobacco ringspot virus and rubbed on cowpea leaves, caused a delay in the appearance of primary virus lesions. This phenomenon is considered to result from the interaction of two materials, an inhibitor which separately decreases and an augmenter which separately increases the number of lesions. The inhibitor was partially purified, and the augmenter was identified indirectly as a soluble oxalate salt. Experiments in which mixtures of partially purified inhibitor, sodium oxalate, and virus were assayed on cowpeas yielded results consonant with the behavior observed with juice. The modes of action of inhibitor and augmenter are discussed.
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