Abstract
Evidence is accumulating that nervous hypothalamic centres may regulate the rate of secretion of corticotrophin. Electrolytic lesions of different parts of the posterior hypothalamus appears to inhibit the release of this hormone (Harris & de Groot, 1950; Hume & Wittenstein, 1950; Porter, 1954; McCann, 1953; Ganong & Hume, 1954; Laqueur et al., 1953). The experiments of Harris & Jacobsohn (1952) and of Jacobsohn (1954) on animals bearing pituitary transplants, the results of Guillemin (1955), Saffran et al. (1955), Porter & Jones (1956) and Porter & Rumsfeld (1956) strongly suggest that a neurohumoral substance from hypothalamic origin is of significance for the secretion of corticotrophin.
Sign-in/Register to access full text options 
Talk to us
Join us for a 30 min session where you can share your feedback and ask us any queries you have
Schedule a callDisclaimer: All third-party content on this website/platform is and will remain the property of their respective owners and is provided on "as is" basis without any warranties, express or implied. Use of third-party content does not indicate any affiliation, sponsorship with or endorsement by them. Any references to third-party content is to identify the corresponding services and shall be considered fair use under The CopyrightLaw.
