Abstract
The acute and chronic swim stress led to an increase in serum dopamine-β-hydroxylase activity in rats. The elevated serum dopamine-β-hydroxylase levels found after stress probably reflect the balance between the rate of release and the rate of disappearance of the enzyme from the circulation. The acute, but not the chronic swim stress resulted in a decrease in dopamine-β-hydroxylase activity in the mesenteric arteries. The changes in dopamine-β-hydroxylase in the arteries following stress probably reflect the balance between the rate of release of the enzyme from the terminals and the rate of replenishment of the enzyme from the cell bodies. The chronic, but not the acute stress resulted in an increase in enzyme activity in the cervical ganglia and in adrenal glands. This finding is consistent with the idea that chronic stress induces the de novo enzyme-protein synthesis.
Sign-in/Register to access full text options 
Talk to us
Join us for a 30 min session where you can share your feedback and ask us any queries you have
Schedule a callDisclaimer: All third-party content on this website/platform is and will remain the property of their respective owners and is provided on "as is" basis without any warranties, express or implied. Use of third-party content does not indicate any affiliation, sponsorship with or endorsement by them. Any references to third-party content is to identify the corresponding services and shall be considered fair use under The CopyrightLaw.
