Abstract
The terms sac and chronic respiratory (CRD) have been widely employed to designate a disease manifested by fibrinous pericarditis, perihepatitis, and thickening of the air sacs. It has been suggested that the disease had a very complex etiology since many agents have been isolated from accessions. Among the more important agents involved are the pathogenic Mycoplasma (also called pleuropneumonia-like organisms or PPLO), infectious bronchitis virus (IBV), Newcastle disease virus (NDV), and certain bacteria most frequently identified as Escherichia coli. This paper reports on methods of reproducing sac experimentally, and makes observations on its probable natural development in the field.
Sign-in/Register to access full text options 
Talk to us
Join us for a 30 min session where you can share your feedback and ask us any queries you have
Schedule a callDisclaimer: All third-party content on this website/platform is and will remain the property of their respective owners and is provided on "as is" basis without any warranties, express or implied. Use of third-party content does not indicate any affiliation, sponsorship with or endorsement by them. Any references to third-party content is to identify the corresponding services and shall be considered fair use under The CopyrightLaw.
