Abstract
AbstractBackgroundNeuroblastoma (NB) is a heterogeneous pediatric solid tumour strongly influenced by epigenetic modifications.MethodsThis review explores the role of N6‐methyladenosine (m6A) modifications and their readers in NB progression.ResultsDysregulation of m6A readers, including YTHDF1/2, YTHDC1, IGF2BP1/2/3, HNRNPA2B1 and HNRNPC, has been associated with susceptibility and progression.ConclusionUnderstanding the crosstalk between m6A readers and NB could offer new insights into diagnosis, prognosis, and treatment strategies.
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