The Course Of Chronic Liver Disease In Patients With Covid-2019 (Literature Review And Own Data)

Muxamedova Z.R

doi:10.37547/tajmspr/volume03issue09-12

Abstract

The pandemic of the new coronavirus COVID-19 has switched medicine around the world on the primary fight against this infection. Patients with chronic liver diseases require increased attention of doctors during an epidemic, since against the background of an exacerbation of their disease, not only the risk of contracting the COVID 19 viral infection increases, but also its more severe course. Patients with confirmed COVID-19 with severe liver damage - high biochemical activity. According to some reports, patients with a severe course of COVID-19 have an increase in ALT levels, a decrease in the number of platelets, a decrease in the level of albumin, and a connection (although not all indicators) with a higher risk of mortality is possible.

Highlights

The global pandemic of the new coronavirus infection (COVID-19) caused by the SARSCoV-2 coronavirus began in Wuhan, China in December 2019 and has since spread around the world
Coronavirus infection (COVID-19) is an acute infectious disease caused by a new strain of the SARS CoV-2 coronavirus with aerosol-capillary and contact-mechanical transmission [2,4,17]
It has been shown that the liver, along with the respiratory tract, is actively participates in the pathological process, and the course and outcome of COVID-19 are closely related to the frequency and extent of liver damage from this infection

Summary

INTRODUCTION

The global pandemic of the new coronavirus infection (COVID-19) caused by the SARSCoV-2 coronavirus began in Wuhan, China in December 2019 and has since spread around the world. Coronavirus infection (COVID-19) is an acute infectious disease caused by a new strain of the SARS CoV-2 coronavirus with aerosol-capillary and contact-mechanical transmission [2,4,17]. The pathogenetic COVID19 is characterized by viremia, local and systemic immune-inflammatory processes, hyperactivity of the coagulation cascade, endothelial disease, hypoxia, which leads to. The development of micro- and macrothrombosis; proceeds from asymptomatic to clinically severe forms with intoxication, fever, damage to the vascular endothelium, lungs, heart, kidneys, gastrointestinal tract, central and peripheral nervous system, system at risk [1,2,3,5,7,9,18,21]

MATERIALS AND METHODS

Findings

CONCLUSION