The ω‐conopeptide SNX‐111, an N‐type calcium channel blocker, dramatically ameliorates brain damage due to transient focal ischaemia

Abstract

Acta Physiologica ScandinavicaVolume 150, Issue 4 p. 459-461 The ω-conopeptide SNX-111, an N-type calcium channel blocker, dramatically ameliorates brain damage due to transient focal ischaemia Q. ZHAO, Q. ZHAO Laboratory for Experimental Brain Research, University of Lund, Lund, SwedenSearch for more papers by this authorM.-L. SMITH, Corresponding Author M.-L. SMITH Laboratory for Experimental Brain Research, University of Lund, Lund, SwedenLaboratory for Experimental Brain Research, Experimental Research Centre, University of Lund, S-221 85 Lund, Sweden.Search for more papers by this authorB. K. SIESJÖ, B. K. SIESJÖ Laboratory for Experimental Brain Research, University of Lund, Lund, SwedenSearch for more papers by this author Q. ZHAO, Q. ZHAO Laboratory for Experimental Brain Research, University of Lund, Lund, SwedenSearch for more papers by this authorM.-L. SMITH, Corresponding Author M.-L. SMITH Laboratory for Experimental Brain Research, University of Lund, Lund, SwedenLaboratory for Experimental Brain Research, Experimental Research Centre, University of Lund, S-221 85 Lund, Sweden.Search for more papers by this authorB. K. SIESJÖ, B. K. SIESJÖ Laboratory for Experimental Brain Research, University of Lund, Lund, SwedenSearch for more papers by this author First published: April 1994 https://doi.org/10.1111/j.1748-1716.1994.tb09713.xCitations: 52AboutPDF ToolsRequest permissionExport citationAdd to favoritesTrack citation ShareShare Give accessShare full text accessShare full-text accessPlease review our Terms and Conditions of Use and check box below to share full-text version of article.I have read and accept the Wiley Online Library Terms and Conditions of UseShareable LinkUse the link below to share a full-text version of this article with your friends and colleagues. Learn more.Copy URL Share a linkShare onFacebookTwitterLinked InRedditWechat Citing Literature Volume150, Issue4April 1994Pages 459-461 RelatedInformation