Abstract
Several theories point to the influence of the geometry of femoral arterial bifurcation in the formation and evolution of atherosclerosis plaques at the level of common, superficial and deep femoral arteries. The objective of this study is to correlate the degrees of calcifications of the femoral tripod with different morphological parameters, namely conicity, tortuosity, proximal and distal diameters of the arteries and angles between the common femoral artery (CFA) and the superficial femoral artery (SFA) and the profound femoral artery (PFA) and the superficial femoral artery. The results showed that only the tortuosity of the common femoral artery influences the formation of atherosclerosis plaque. These results allow us to predict the degree of calcification of the femoral tripod based on the tortuosity of the common femoral artery.
Highlights
Arterial calcifications are clearly related to mortality
The objective of this study is to correlate the degrees of calcifications of the femoral tripod with different morphological parameters, namely conicity, tortuosity, proximal and distal diameters of the arteries and angles between the common femoral artery (CFA) and the superficial femoral artery (SFA) and the profound femoral artery (PFA) and the superficial femoral artery
The results showed that only the tortuosity of the common femoral artery influences the formation of atherosclerosis plaque
Summary
Arterial calcifications are clearly related to mortality. After adjustment for age, dialysis, gender, ethnicity, diabetes, non-dialysis renal failure, hypertension, smoking, parathyroid surgery, body mass index, medial and intimal calcification increase the relative risk of mortality by 5 and 12 times compared to patients without calcification [2]. Calcifications alter the morphology and compliance of the arterial wall and reduce the effectiveness of angioplasty and stenting [3]. Technical success and permeability are altered in cases of severe arterial calcifications [4] by increasing the risk of arterial dissection and recoil [3] after balloon angioplasty and by promoting under-expansion, malposition and fracture of stents [5] [6]. The aim of this work is to evaluate the influence of a few morphological parameters on atherogenesis
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