Abstract
A low dose of sufentanil was insufficient to suppress endotracheal intubation (ETI) induced cardiovascular response. This study investigated the effects of combined sufentanil and remifentanil on hemodynamic changes during anesthetic induction and ETI.
Highlights
A low dose of sufentanil was insufficient to suppress endotracheal intubation (ETI) induced cardiovascular response
A low dose of sufentanil combined with propofol is inadequate to well suppress ETI induced increase of heart rate (HR) and blood pressure (BP) [4]
The hemodynamic profiles of the two groups showed that the cardiovascular responses after ETI in Group R were less fluctuated than in Group S
Summary
A low dose of sufentanil was insufficient to suppress endotracheal intubation (ETI) induced cardiovascular response. This study investigated the effects of combined sufentanil and remifentanil on hemodynamic changes during anesthetic induction and ETI. Anesthetic induction and endotracheal intubation (ETI) can cause hemodynamic fluctuation. Opioids in combination with propofol are widely used for suppressing ETI induced cardiovascular response during anesthetic induction [3]. Sufentanil, an analog of fentanyl, is a proper opioid for anesthetic induction because of less impact on hemodynamics. A previous study showed that sufentanil dose-dependently suppressed ETI induced cardiovascular response [4]. A low dose of sufentanil combined with propofol is inadequate to well suppress ETI induced increase of heart rate (HR) and blood pressure (BP) [4]. A larger dose of sufentanil may Accepted: September 27, 2021
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