Abstract
Miscarriage and fetal growth retardation which cause alterations in the protein synthesis function of the uteroplacental complex occupy one of the leading positions in the structure of causes of perinatal and child morbidity and mortality. Investigation of the dynamics of TBG, AMGF, PAMG levels during treatment of uteroplacental insufficiency was conducted. The changes revealed in the protein synthesis indicate dysaptation, which is more evident if there is fetal growth retardation than if there is threatened miscarriage.
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