Abstract
The atopic march described the progression from atopic dermatitis (AD) toward asthma and allergic rhinitis,1 variably interpreted initially as AD and later with the onset of wheezing or rhinitis, or as all 3 atopic disorders in the order of AD, then asthma, then rhinitis. A central role for AD barrier dysfunction was implicated by the development of airway sensitization in tape-stripped murine models sensitized epicutaneously. Data-driven assessments, however, suggest a more complex, multimorbidity relationship among these atopic disorders, reflecting their shared pathomechanisms, environmental triggers, and genetic risk factors.
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