Abstract
The analysis of associations between cytokine network genes and inverse co-morbidity of bronchial asthma and tuberculosis
Highlights
Co-morbidity, a simultaneous development of several pathologic conditions in one individual, is a common phenomenon in clinical practice [1]
According to the concept of syntropy/dystropy, an accumulation of co-morbidity in families suggest a non-random nature in the combination of the diseases in individuals and their relatives and, the importance of genes, called “syntropic genes”, which are responsible for their simultaneous development [,3]
All the polymorphisms were present in accordance with the expectation under Hardy-Weinberg equilibrium except for the rs525891 of TNFRSF1B (р = 0.014)
Summary
Co-morbidity, a simultaneous development of several pathologic conditions in one individual, is a common phenomenon in clinical practice [1]. The issue of co-morbidity is a specific subject of genetic studies. Various factors can participate in the suppression of one disease in the context of the development of another, including so-called dystropic genes [2].
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