Abstract
Microsomal monooxygenase activities toward a number of model substrates, i.e., 7-methoxy- and 7-ethoxyresorufin and 7-ethoxycoumarin, were from 7- to 28-fold increased in a teflubenzuron-resistant field strain (resistance ratio RR = 31) and in two laboratory-selected teflubenzuron-resistant strains (RR = 18 and 36) of the diamondback moth as compared with a susceptible strain of this insect pest of cruciferous crops. No 7-pentoxyresorufin O-depentylase activity was detected in any of the strains. Aldrin epoxidase activity varied up to 3-fold among the strains studied. Because there is no cross-resistance from pyrethroid resistance to teflubenzuron and because pyrethroid resistance has been attributed to increased microsomal monooxygenase activity as well, we propose that different forms of cytochrome P450s are involved in the detoxication of pyrethroids and teflubenzuron in diamondback moth larvae.
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