Abstract
Chronic obstructive pulmonary disease (COPD) is a heterogeneous disease with significant between-patient differences in symptoms, severity of airflow limitation, worsening of lung function, frequency of exacerbations, comorbidities and prognosis.1 Although COPD is characterised by neutrophilic chronic airway inflammation, findings from several studies of (induced) sputum and bronchial biopsies2,3 have indicated the presence of eosinophilic airway inflammation in a subgroup of patients, either with stable disease or during acute exacerbations of COPD (AECOPD).
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