Abstract
Background and Aims : Hyperactive poly(ADP-ribose) polymerases (PARP) promote ischemic heart failure (IHF) after myocardial infarction (MI). However, the role of Tankyrases (TNKSs), members of PARP family, in the progression of IHF remains unknown. We aimed to explore the role of TNKSs in pathophysiological process of IHF and the therapeutic potential of TNKS inhibition for IHF.
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