Abstract
The norepinephrine transporter gene (SLC6A2) and movement time deficits have been consistently reported to be associated with attention-deficit hyperactivity disorder (ADHD). This study aimed to examine whether the SLC6A2 rs36011 (T)/rs1566652 (G) haplotype affected white matter (WM) structural property in children with ADHD and whether those alterations were associated with movement time deficits.
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