Abstract
Basal ganglia circuitry, organized in largely parallel dorsal and ventral/limbic pathways, is modulated by dopamine. We recently reported cortico-basal ganglia intrinsic functional connectivity (iFC) alterations in schizophrenia and identified decreased striatal dopamine synthesis capacity (DSC) in patients with schizophrenia during symptomatic remission of positive symptoms. However, the functional relationship between altered within-basal ganglia iFC and striatal-DSC is unknown in schizophrenia.
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