Abstract
Biomarkers in cognitively vulnerable populations like those with coronary artery disease (CAD) may inform earlier intervention in vascular neurodegeneration. Circulating ceramide C18:0 (CerC18:0) is associated with changes in verbal memory in early neurodegeneration and CAD progression. We hypothesized that plasma CerC18:0 and its accumulation in biosynthesis pathways (catabolism of sphingomyelin (SM), salvage from monohexosylceramides (MHx), formation from sphingosine-1-phosphate (S1P)) would be associated with declining verbal memory performance in CAD.
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