Abstract
Accumulating data indicate that abnormal glutamatergic signaling plays an important role in the pathophysiology of schizophrenia. RNA editing is a key developmental process in the brain that regulates the trafficking of AMPA glutamate receptors to the synapse, a process that is impaired in aging. In this study, we have tested the hypothesis that GluA2-AMPA receptor RNA editing is abnormal in schizophrenia.
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