Abstract
We have recently demonstrated that vulnerability to relapse of fear is related to individual differences in the rate of extinction (with slow extinguishers being more vulnerable). Here we examined the molecular basis for these individual differences in rate of extinction and found differences between “fast” and “slow” extinguishers in NMDA receptor protein levels. We then examined whether this translated to differences in responding to an NMDA receptor partial-agonist, D-cycloserine (DCS).
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