Abstract
BACKGROUND: Young adults who were born prematurely have higher rates of heart failure, cardiac hypertrophy, and systolic dysfunction than those born full term. The mechanism underlying these abnormalities likely include changes in the contractile machinery of the heart, including α myosin heavy chain (αMHC) and β myosin heavy chain (βMHC). microRNA-208a and microRNA-208b play key roles in the regulation of myosin heavy chain expression. In adult mice, previous exposure to perinatal inflammation and postnatal …
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