Abstract
MANY clinical and experimental studies have been performed to study the etiology, diagnosis and treatment of traumatic fat embolism. Recently, a less well understood counterpart, termed "nontraumatic fat embolism," has been recognized. In documenting the first case of pulmonary and systemic fat embolism as a complication of hypercortisonism in man, Hill,1 in 1961, suggested that corticosteroids administered in high dosages were a major factor in the evolution of systemic fat emboli.Another case of fat embolism as a possible complication of hypercortisonism is reported. This potentially fatal entity is particularly important because of the increasing interest in homotransplantation and the . . .
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