Abstract
Background: The vessel stenosis and re-stenosis processes are mainly related to cellular and molecular events. The vascular smooth muscle cell (VSMC) activation is the basic element in these lesions. Based on monocyte and macrophage inflammatory responses and also VSMC motility, a complex protein network is dynamically proposed between these cells. The aim of this study was to highlight the protein communications involved in VSMC proliferation and migration signaling pathways. Keywords: Atherosclerosis, network, cluster, pathway, VSMC, macrophage, monocyte.
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