Abstract
RATIONALE: Epigenetic control of gene expression profiles is a ubiquitous mechanism during cell differentiation, organogenesis or chronic inflammatory reactions. Recent studies showed that allergen exposure during pregnancy increases bronchial hypersensitivity of their offspring in a murine model of bronchial asthma, however, no such phenomena were reported in humans. In the present study, the role of epigenetic control in the onset of allergic diseases was investigated.
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