Abstract
We read with great interest the article by Mieth and colleagues1 dealing with the effect of the benzothiazepine-type calcium channel blocker, diltiazem, on aortic aneurysm formation in a mouse model. The results of their study demonstrated that diltiazem significantly prevented abdominal aneurysm formation in a blood pressure–independent manner in male apolipoprotein E–deficient mice infused with angiotensin II. In addition, the authors showed that diltiazem inhibited the interleukin-6 (IL-6)–induced mRNA expression of IL-1β and the monocyte chemoattractant protein …
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