Abstract
The role of the sympathetic nervous system in the pathogenesis of essential human hypertension is not yet established [6, 7, 10, 11, 25, 36]. In spite of this the majority of drugs currently used in the treatment of human hypertension act by modifying peripheral and/or central adrenergic mechanisms [27]. Whereas the site and mode of action is relatively well understood in the peripheral sympathetic nervous system this is much less the case for centrally acting drugs.
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