Abstract
In non-diabetic rats, the mitochondrial phosphorylative activity per unit of cytochrome a(+a 3) in the remnant liver 3 hours after hepatectomy increased to approximately 140% of sham-operated controls without significant decrease in the energy charge. In diabetic rats the enhancement in mitochondrial phosphorylative activity following hepatectomy was inhibited in proportion to the severity of impaired insulin secretion, with a concomitant decrease in hepatic energy charge. It is suggested that insulin plays an important role in initiating an enhancement of mitochondrial oxidative phosphorylation.
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