Abstract

Objective: Superior semicircular canal dehiscence (SSCD) syndrome has been defined as the absence of bone overlying the superior semicircular canal facing toward the dura of the middle cranial fossa. This bony defect creates a direct communication between the dura and labyrinthine membranous structure, leading to disruption of physiological pressure gradient within the membranous structures and acts as a mobile third window. SSCD patients present hyperacusis, autophony, disequilibrium, and sound- or pressure-induced vertigo. Apart from the avoidance of aggravating factors, there is no effective medical treatment for SSCD. Therefore, surgical management aims to eliminate the communication between the dura and inner ear structures either by reconstructing the skull base or sealing the bony defect over the petrous bone.

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