Abstract
Malignant hypertensive nephrosclerosis develops in some but not all cases of severe hypertension; the underlying molecular mechanisms are not clear. We compared gene expression in renovascular hypertensive rats which developed malignant hypertensive nephrosclerosis with gene expression in rats which were hypertensive but did not develop malignant nephrosclerosis. Further, we investigated the potential role of one candidate molecule identified from our gene expression screen, Interleukin-11 (IL-11).
Full Text 
Sign-in/Register to access full text options
Sign-in/Register to access full text options 
Published version (
Free)
Free) 
Talk to us
Join us for a 30 min session where you can share your feedback and ask us any queries you have
Schedule a call
