Abstract
This article uses a biologic model of sudden cardiac death to identify structural abnormalities that serve as the substrate for sustained arrhythmias and functional changes that are transient and necessary for triggering an arrhythmia. The biologic framework is a valuable tool to use in understanding the relationship between structure and function and in organizing patients' clinical presentations and outcomes.
Full Text 
Sign-in/Register to access full text options
Sign-in/Register to access full text options 
Paper version not known
Talk to us
Join us for a 30 min session where you can share your feedback and ask us any queries you have
Schedule a call
