Abstract
Neurological complications occur in approximately 25% ofpatients with WM. Although they are most often present as pe-ripheral neuropathy, hyperviscosity can produce central nervoussystem (CNS) changes that resolve promptly after plasma exchange.Rarely is the CNS directly involved as is the case in Bing-Neelsyndrome (BNS), which results from infiltration of pons, medulla,periventricular white matter, or leptomeningeal spaces by lympho-plasmacytoid cells.
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