Abstract

Successful treatment of an extensive retinal damage due to blunt ocular trauma

Highlights

  • Ocular blunt trauma can cause massive submacular hemorrhage (SMH) which results in suddenly deterioration of visual acuity (VA) and permanent visual loss

  • In posterior segment evaluation post-injection month 1, we observed that intravitreal perfluoropropane gas completely resorbed, VA improved to 20/25, minimal submacular coagulum, parafoveal pigmentation, large choroidal rupture region involved to only parafoveal region sparing to fovea, minimal subretinal arc shaped hemorrhage on superior hemisphere and laser scars around the retinal tear

  • In 1-year follow-up period, VA was still remained 20/25 and we considered that choroidal rupture did not involve the fovea and intravitreal injection of bevacizumab may additional effect for resorbing the displaced SMH and prevent secondary choroidal neo-vascular membrane (CNM) development which may involve to central fovea

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Summary

Introduction

Ocular blunt trauma can cause massive submacular hemorrhage (SMH) which results in suddenly deterioration of visual acuity (VA) and permanent visual loss. The most common treatment strategy is to displace of hemorrhage with expansible gas injection [3] In this case report, we presented traumatic peripheral retinal tears and SMH which was completely displaced by intravitreal pure perfluoropropane gas (C3F8) injection. In posterior segment evaluation post-injection month 1, we observed that intravitreal perfluoropropane gas completely resorbed, VA improved to 20/25, minimal submacular coagulum, parafoveal pigmentation, large choroidal rupture region involved to only parafoveal region sparing to fovea, minimal subretinal arc shaped hemorrhage on superior hemisphere and laser scars around the retinal tear. In 1-year follow-up period, VA was still remained 20/25 and we considered that choroidal rupture did not involve the fovea and intravitreal injection of bevacizumab may additional effect for resorbing the displaced SMH and prevent secondary CNM development which may involve to central fovea.

Discussion
Findings
13. Ophthalmology 111
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