Abstract
Hemiballism with corresponding striatal T1 hyperintensity on MR imaging has occasionally been reported in patients with nonketotic hyperglycemia. However, the subthalamic nucleus lesion, which is believed to be pathogenetically related to hemiballism, is rarely documented in a living patient with nonketotic hyperglycemia. We describe a patient with nonketotic hyperglycemia-induced hemiballism, whose responsible lesion (ie, the subthalamus) was demonstrated by MR imaging.
Highlights
Hemiballism-hemichorea (HB-HC) is an involuntary, irregular, wide-amplitude, and poorly patterned movement disorder
MR imaging revealed the sole involvement of the contralateral subthalamic nucleus (STN) without a putaminal lesion
Subthalamic involvement has never been reported in hyperglycemia-induced hemiballism, acute hemiballism was reported in a small cavernous angioma or infarction confined to the STN.[10,11]
Summary
Hemiballism-hemichorea (HB-HC) is an involuntary, irregular, wide-amplitude, and poorly patterned movement disorder. There are only scanty reports documenting the subthalamic involvement of nonketotic hyperglycemia in a living patient, which prompted us to report a case of nonketotic hyperglycemia-induced hemiballism, with the responsible subthalamic lesion confirmed by MR imaging
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