Abstract
It is well known that zinc has important biological functions. Clinical manifestations in zinc-deficient animals include growth retardation, testicular atrophy, skin changes, and poor appetite. On the other hand, high levels of dietary zinc have been shown to induce copper deficiency in rats and to interfere with the metabolism of calcium and iron. Little is known on the oral toxicity of zinc in mammals. However, some toxic effects in human subjects, rodents, and sheep have been reported. In order to extend the information about the oral toxicity of zinc, a semichronic toxicity study of zinc acetate in rats has been carried out in this paper.
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