Year-end Sale % OFF 
Abstract
A marked depletion of hepatic glycogen, an appreciable decrease in the hepatic glycogenesis, and a lowered glucose tolerance in tuberculous guinea pigs were demonstrated. Incorporation of glycine-1-C 14 into hepatic glycogen in vivo showed a tendency to increase as a result of experimental tuberculosis. Depletion of hepatic glycogen in experimental tuberculosis has been ascribed to the suppressed glycogenesis.
Talk to us
Join us for a 30 min session where you can share your feedback and ask us any queries you have
Schedule a callDisclaimer: All third-party content on this website/platform is and will remain the property of their respective owners and is provided on "as is" basis without any warranties, express or implied. Use of third-party content does not indicate any affiliation, sponsorship with or endorsement by them. Any references to third-party content is to identify the corresponding services and shall be considered fair use under The CopyrightLaw.
