Abstract
1. In embryonic chick liver glycogen mobilization proceeds parallel to the hatching process. The glycogen stores are degraded earlier than normal when the hatching process is induced by L-thyroxine, and at a later stage of incubation when the period of incubation is prolonged by thiourea. 2. In untreated chicks glycogenolysis does not seem to be mediated by cyclic AMP (cAMP), although glycogen metabolism responds to changes of cAMP levels caused by exogenous glucagon. 3. Similar to glycogenolysis, ATP concentrations decrease towards the end of incubation. The fall in ATP is accelerated in L-thyroxine treated embryos. 4. Due to the limited O 2 supply, it seems possible that the decrease ofglycogen and ATP before hatching is released by hypoxia.
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