Abstract
Voluntary fasting as treatment for severe obesity will induce the loss of body water, minerals, protein, and fat. Excretion of hydroxyproline and, to a lesser degree, calcium increases as the duration of the fast is prolonged. The increased urinary hydroxyproline excretion reflects in part the skeleton's contribution to the buffering of the induced metabolic acidosis and to a greater degree the derivation of substrate for gluconeogenesis from collagen rich extracellular reserves such as skin. The data presented also reveal that young adults catabolize collagen as a metabolic reserve for gluconeogenesis to a much greater degree than do older adults.
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