Abstract
SummaryCyclic AMP and other agents capable of elevating cyclic AMP levels were incubated with human blood cells to test ability to inhibit unsaturated vitamin B12 binding protein release. Except for NaF, none of the agents tested showed a significant effect on UBBC increment at 24 hr compared to controls. As shown previously, NaF fully prevented UBBC release, even in the presence of added Li+. From these data, it is concluded that release of unsaturated vitamin B12 binding protein from human blood cells is not mediated primarily through changes in intracellular cyclic AMP levels.
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Schedule a callMore From: Proceedings of the Society for Experimental Biology and Medicine. Society for Experimental Biology and Medicine (New York, N.Y.)
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