Abstract
Previous studies have demonstrated that phlebosclerosis follows prolonged venous hypertension secondary to congestive failure. Thus far this has been shown in the hepatic veins, the portal vein, the inferior vena cava, the pulmonary veins and the splenic vein. The coronary vein has a remarkable immunity in this respect, even in cases of unusually prolonged congestive failure. This is explained by a free anastomoses between the coronary and the Thebesian veins aided by the usual incompetence of the Thebesian valve in the coronary sinus. The pressure within the coronary vein cannot, therefore, attain a sufficient height to produce phlebosclerosis. This immunity offers a cogent support to the causative relation between intravascular pressure and phlebosclerosis and indirectly throws light on its counterpart, arteriosclerosis.
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