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Abstract
Development of insulin-dependent diabetes mellitus in children is accompanied by essential disturbances in the peripheral subdivision of the erythron system, manifested in enhanced cell polymorphism, imbalance in the composition of membrane phospholipids and their increased microviscosity, reduced lipoprotein concentration, and enhanced reversible aggregation. These disturbances are most pronounced during metabolic decompensation accompanied by ketoacidosis, which agrees with the hypothesis on their role in the development of vascular complications.
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