Abstract
In studies on the monthly rat pups who had undergone by intrauterine experimental hypoxia of varying severity was shown that severe hypoxia causes in lung tissue hyperhydration of air-blood barrier and expressed mitochondrial dysfunction (MD). In the myocardium, together with a thickening of histogematic barrier, in the mitochondrial apparatus of cardiomyocytes besides signs of MD occurs the formation of certain compensatory-adaptive mechanisms to maintaining an adequate energy supply of the heart. After hypoxia of average degree hyperhydration of biological barriers was absence, in the mitochondrial apparatus of the heart and lungs tissues in the absence of MD was formed some compensatory mechanisms aimed at preventing of tissue hypoxia.
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