Abstract
卒中相关性肺炎（SAP）是急性缺血性卒中的主要并发症，与患者的病死率和医疗费用增加密切相关。发生SAP的病理生理学机制可能与误吸及卒中诱导的免疫抑制有关，后者是在卒中发生后通过复杂的循环性和神经性通路包括下丘脑-垂体-肾上腺轴、交感/副交感神经系统而诱发。.
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