Abstract
Statins are the cornerstone for lowering low-density lipoprotein cholesterol (LDL-C) for cardiovascular disease (CVD) prevention. Moreover, as reported recently, the benefits of statin therapy extend similarly to men and women [1]. While statins are safe and well tolerated, like all treatments, a proportion of patients report side effects. Muscle symptoms are the most prevalent side effects reported with statin therapy, and one of the main reasons for non-adherence or discontinuation of treatment. While it is recognised that statins do cause a rare side effect known as myositis, defined as muscle symptoms in association with a substantially elevated serum creatine kinase [CK] concentration, most statin-associated muscle symptoms (SAMS) are not accompanied by marked CK elevation.
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