Abstract
THE increased concentration of plasma amylase during chronic renal insufficiency has long been recognized and is considered to be a result of renal excretory failure. Plasma amylase is reported to rise in parallel with urea concentration1 but not to heights encountered in acute pancreatitis.2 Similar rises in amylase might be expected to accompany urea retention in acute renal insufficiency, but this phenomenon has received little attention.The clinical interest in plasma amylase elevation during acute renal insufficiency is heightened by the occurrence in this condition of acute abdominal crises that may resemble acute pancreatitis. The decision to perform or defer . . .
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