Abstract
Some of the factors controlling tissue growth in animals and cell proliferation in unicellular organisms are reviewed. Radiation damage in diploid yeast cells manifests itself sometimes in delayed lethal effect and decreased cell division rate over several generations in the progeny of the irradiated cell. Recovery of the progeny of irradiated yeast cells from phenotypic expression of radiation damage can be delayed over many generations and is similar, in time rate of onset to the onset of radiation induced animal tumors. A parallel suggests itself between the mechanism of somatic-radiation carcinogenesis and delayed recovery of unicellular organisms from radiation effect. (auth)
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