Abstract
THYROTOXICOSIS in the prepubertal patient is almost invariably due to Graves disease. The recent report of Popma et al<sup>1</sup>underscores, however, the responsibility of the solitary hyperfunctioning nodule for clinically significant hyperthyroidism in the pediatric patient. To our knowledge, this communication presents the second chemically documented example of hyperthyroidism due to a benign solitary hyperfunctioning adenoma in a child, and the first to produce both excessive thyroxine (T<sub>4</sub>) and triiodothyronine (T<sub>3</sub>) levels in the face of a normal serum thyroid-stimulating hormone (TSH) level. <h3>Report of a Case</h3> An 11 1/2-year-old girl was first seen in referral during December 1972. A previous physical examination in April 1971 had been normal. In February 1972, her pediatrician noted a right thyroid nodule. History disclosed some increase in sweating and a tendency to use fewer blankets at night. Her mother observed an increased irritability and a preference for the family room
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